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Atypical Diabetes: Pathophysiology, Clinical Presentations, and Treatment Options by Boris Draznin Although the vast majority of diabetes cases (type 1 and type 2) are polygenic diseases, research, funded by the American Diabetes Association, has found that some forms of diabetes, distinct from type 1 or type 2 diabetes, are caused by single gene mutations. It has been estimated that these monogenic forms of diabetes account for up to five percent of all diabetes cases. In addition to monogenic diabetes, there are other atypical causes of diabetes, including: genetic defects in insulin action; diseases of the exocrine pancreas; and endocrinopathies. Given recent advances in our understanding of sub-types of diabetes, the time has come for a book on how to correctly diagnose and treat patients with atypical forms of this disorder. The book will be divided into three parts, each exploring distinct categories of atypical diabetes. Part I: Monogenic Diabetes: Genetic Defects of β-cell development and function; Part II: Insulin resistance, genetic defects in insulin action, and diseases of exocrine pancreas; Part III: Endocrinopathies, Immune-mediated diabetes, Diabetes of Unknown cause, and Other genetic syndromes sometimes associated with diabetes.

Atypical Diabetes: Pathophysiology, Clinical Presentations, and
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Diabetes in African Americans | Postgraduate Medical Journal
African Americans have a high rate of diabetic complications, because of poor glycaemic control and racial disparities in health care in the USA. African Americans with diabetes may have an atypical presentation that simulates type 1diabetes, but then their subsequent clinical course is typical of type 2 diabetes. Culturally 
Adult-Onset Atypical (Type 1) Diabetes - Semantic Scholar
autoantibodies and fluctuating insulinopenia). RESEARCH DESIGN ANDMETHODS — Eight patients with atypical type 1 diabetes ments and ketosis- prone episodes, as well as clinical features of type 2 diabetes. Only one subgroup could be strictly Diabetes Care 27:1108 –1114, 2004. In 1997, the American Diabetes 
Updates in diabetic peripheral neuropathy - NCBI - NIH
Similar to DPN, the neuropathy of TIND generally follows a length-dependent pattern, but, in contrast, the pain and autonomic symptoms are more extensive and less responsive to opioids. The underlying pathophysiology is poorly understood, although it has been suggested that rapid glycemic control both 
Hyperosmolar Hyperglycemic State Clinical Presentation: History
Hyperosmolar hyperglycemic state (HHS) is 1 of 2 serious metabolic derangements that occurs in patients with diabetes mellitus (DM) and can be a life-threatening emergency. It is less common Other common symptoms include nausea, vomiting, weakness, lethargy, and muscle cramps. They do not 
What type of diabetes does my patient have and is it relevant? - AJOL
mellitus were given names descriptive of their clinical presentation. These are still in wide use even today: insulin-dependent diabetes mellitus (IDDM) and non- insulin-dependent diabetes mellitus. (NIDDM). However, as insight intopathogenesis and treatment strategies evolved, it became difficult to correctly classify 
Syndromes of ketosis-prone diabetes mellitus - UpToDate
In this review, we describe the current state of knowledge in regard to the natural history, pathophysiology, and treatment of the subgroups of KPD, with an METHODS In this cross-sectional analysis, we reviewed clinical charts of type 2diabetic patients with DKA admitted to Cayetano Heredia Hospital between 2001 and 
Syndromes of Ketosis-Prone Diabetes Mellitus - NCBI - NIH
Recent data from longitudinally followed cohorts have clarified the clinicalfeatures and taxonomy of some of these syndromes that are “intermediate” have taken advantage of the striking and unequivocal presentation of DKA in “atypical” patients to define syndromes of ketosis-prone diabetes (KPD).